The breakdown and release of hyaluronan (HA) from the extracellular matrix has been hypothesized to act as an endogenous signal of injury. To test this hypothesis, we generated mice that conditionally overexpressed human hyaluronidase 1 (
Jun Muto, Yasuhide Morioka, Kenshi Yamasaki, Margaret Kim, Andrea Garcia, Aaron F. Carlin, Ajit Varki, Richard L. Gallo
The Editorial Board will only consider comments that are deemed relevant and of interest to readers. The Journal will not post data that have not been subjected to peer review; or a comment that is essentially a reiteration of another comment.