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Usage Information

Erythropoietin: when liability becomes asset in neurovascular repair
Maria B. Grant, … , Michael E. Boulton, Alexander V. Ljubimov
Maria B. Grant, … , Michael E. Boulton, Alexander V. Ljubimov
Published January 24, 2008
Citation Information: J Clin Invest. 2008;118(2):467-470. https://doi.org/10.1172/JCI34643.
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Commentary

Erythropoietin: when liability becomes asset in neurovascular repair

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Abstract

Erythropoietin (Epo) leads to the proliferation and differentiation of erythroid precursors, but is also involved in diverse nonhematopoietic biological functions. In this issue of the JCI, Chen, Smith, and colleagues demonstrate that the temporal expression of Epo is critical for determining whether physiological or pathological repair occurs following neurovascular retinal injury in the oxygen-induced retinopathy neonatal mouse model (see the related article beginning on page 526). The pleiotrophic properties of Epo make it a likely novel therapy for treatment of neurovascular damage, but the timing of its use must be carefully considered to prevent untoward effects.

Authors

Maria B. Grant, Michael E. Boulton, Alexander V. Ljubimov

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Usage data is cumulative from May 2024 through May 2025.

Usage JCI PMC
Text version 327 17
PDF 61 12
Figure 95 3
Citation downloads 66 0
Totals 549 32
Total Views 581
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Usage information is collected from two different sources: this site (JCI) and Pubmed Central (PMC). JCI information (compiled daily) shows human readership based on methods we employ to screen out robotic usage. PMC information (aggregated monthly) is also similarly screened of robotic usage.

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