The mammalian heart contains a population of resident macrophages that expands in response to myocardial infarction through the recruitment of monocytes. Infarct macrophages exhibit high phenotypic diversity and respond to microenvironmental cues by altering their functional properties and secretory profile. In this issue of the
Nikolaos G. Frangogiannis
Title and authors | Publication | Year |
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Fibrosis of the diabetic heart: Clinical significance, molecular mechanisms, and therapeutic opportunities
I Tuleta, NG Frangogiannis |
Advanced Drug Delivery Reviews | 2021 |
Role of Podoplanin-Positive Cells in Cardiac Fibrosis and Angiogenesis After Ischemia
M Cimini, R Kishore |
Frontiers in physiology | 2021 |
A double-edged sword of immuno-microenvironment in cardiac homeostasis and injury repair
K Sun, Y Li, J Jin |
Signal Transduction and Targeted Therapy | 2021 |
Bioinformatics and Immune Infiltration Analyses Reveal the Key Pathway and Immune Cells in the Pathogenesis of Hypertrophic Cardiomyopathy
XZ Zhang, S Zhang, TT Tang, X Cheng |
Frontiers in Cardiovascular Medicine | 2021 |
Crosstalk Between Cardiac Cells and Macrophages Postmyocardial Infarction: Insights from In Vitro Studies
P Hitscherich, E Lee |
Tissue Engineering Part B: Reviews | 2021 |