The mammalian heart contains a population of resident macrophages that expands in response to myocardial infarction through the recruitment of monocytes. Infarct macrophages exhibit high phenotypic diversity and respond to microenvironmental cues by altering their functional properties and secretory profile. In this issue of the
Nikolaos G. Frangogiannis
Title and authors | Publication | Year |
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Loss of Macrophage Wnt Secretion Improves Remodeling and Function After Myocardial Infarction in Mice
D Palevski, LP LevinKotler, D Kain, N NaftaliShani, N Landa, T BenMordechai, T Konfino, R Holbova, N Molotski, R RosinArbesfeld, RA Lang, J Leor |
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease | 2017 |
Possible Muscle Repair in the Human Cardiovascular System
L Sommese, A Zullo, C Schiano, FP Mancini, C Napoli |
Stem Cell Reviews and Reports | 2017 |
Mechanisms of cardioprotection via modulation of the immune response
Grilo GA, Shaver PR, de Castro Brás LE |
Current Opinion in Pharmacology | 2017 |