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Abstract
Hepatitis A virus (HAV) usually results in asymptomatic infection or mild disease, but in a small subset of patients it can lead to severe disease and even liver failure. In this issue of the JCI, Kim et al. identify a polymorphism associated with this increased disease risk in the gene that encodes the HAV receptor — TIM1. Importantly, this TIM1 gene polymorphism protects from the development of atopic diseases in HAV-seropositive individuals.
Authors
Lucienne Chatenoud, Jean-François Bach
Citation information
Citations to this article (5)
| Title and authors | Publication | Year |
|---|---|---|
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HAVCR1 Affects the MEK/ERK Pathway in Gastric Adenocarcinomas and Influences Tumor Progression and Patient Outcome
J Xue, Y Li, J Yi, H Jiang |
Gastroenterology Research and Practice | 2019 |
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T-helper 17-related cytokines and IgE antibodies during hepatitis A virus infection in children
JL Trujillo-Ochoa, KF Corral-Jara, G Escobedo-Meléndez, M Realpe, A Panduro, S Roman, NA Fierro |
Memórias do Instituto Oswaldo Cruz | 2015 |
|
Rethinking the immune properties of bilirubin in viral hepatitis: from bench to bedside
KF Corral-Jara, JL Trujillo-Ochoa, M Realpe, A Panduro, S Roman, NA Fierro |
IBMS BoneKEy | 2015 |
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The Hygiene Hypothesis: An Explanation for the Increased Frequency of Insulin-Dependent Diabetes
JF Bach, L Chatenoud |
Cold Spring Harbor Perspectives in Medicine | 2012 |
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Pulmonary surfactant: an immunological perspective
ZC Chroneos, Z Sever-Chroneos, VL Shepherd |
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology | 2009 |
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