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Samuel Klein
Published June 1, 2004
Citation Information: J Clin Invest. 2004;113(11):1530-1532. https://doi.org/10.1172/JCI22028.
Citation Information: J Clin Invest. 2004;113(11):1530-1532. https://doi.org/10.1172/JCI22028.
Abstract
Increased plasma fatty acid concentrations may be responsible for many of the metabolic abnormalities associated with abdominal obesity. Excessive visceral fat is associated with insulin resistance and other metabolic risk factors for coronary heart disease. A study reported in this issue of the JCI evaluates the relative contribution of fatty acids released during lipolysis of visceral adipose tissue triglycerides to portal and systemic fatty acid flux in human subjects.
Authors
Samuel Klein
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Citations to this article in year 2007 (2)
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|---|---|---|
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The role of skeletal muscle insulin resistance in the pathogenesis of the metabolic syndrome
KF Petersen, S Dufour, DB Savage, S Bilz, G Solomon, S Yonemitsu, GW Cline, D Befroy, L Zemany, BB Kahn, X Papademetris, DL Rothman, GI Shulman |
Proceedings of the National Academy of Sciences | 2007 |
|
HIV-associated adipose redistribution syndrome (HARS): etiology and pathophysiological mechanisms
Lichtenstein K, Balasubramanyam A, Sekhar R, Freedland E |
AIDS Research and Therapy | 2007 |
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