Impaired glucose phosphorylation and transport in skeletal muscle cause insulin resistance in HIV-1–infected patients with lipodystrophy
Georg M.N. Behrens, … , Georg Brabant, Reinhold E. Schmidt
Georg M.N. Behrens, … , Georg Brabant, Reinhold E. Schmidt
Published November 1, 2002
Citation Information: J Clin Invest. 2002;110(9):1319-1327. https://doi.org/10.1172/JCI15626.
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Categories: Article Metabolism

Impaired glucose phosphorylation and transport in skeletal muscle cause insulin resistance in HIV-1–infected patients with lipodystrophy

Abstract

Research Article

Authors

Georg M.N. Behrens, Anne-Rose Boerner, Klaus Weber, Joerg van den Hoff, Johann Ockenga, Georg Brabant, Reinhold E. Schmidt

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Figure 1

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Three-compartment model for the F-18-FDG kinetics of the PET data. Dynam...
Three-compartment model for the F-18-FDG kinetics of the PET data. Dynamically acquired PET data and arterial plasma time course of F-18-FDG activity were used as input functions in the compartmental modeling. The rate constants represent inward transport (k1), outward transport (k2), and phosphorylation (k3) of F-18-FDG.