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Citations to this article

Increased glycosphingolipid excretion associated with proteinuria.
R R Townsend, … , S C Li, Y T Li
R R Townsend, … , S C Li, Y T Li
Published July 1, 1978
Citation Information: J Clin Invest. 1978;62(1):119-123. https://doi.org/10.1172/JCI109095.
View: Text | PDF
Research Article

Increased glycosphingolipid excretion associated with proteinuria.

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Abstract

The urine of patients with proteinuria of various etiologies was examined to determine if proteinuria alone was associated with significant glycosphingolipiduria. In all cases of proteinuria examined, the level of glycosphingolipids in the urine was found to be markedly elevated. There was no evidence of a glycosphingolipid storage disorder in any case. It was concluded that significant glycosphingolipiduria may occur in proteinuria as well as in the glycosphingolipid storage disorders.

Authors

R R Townsend, R M Orth, C M Clawson, S C Li, Y T Li

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Total citations by year

Year: 2021 2014 2013 1985 1982 Total
Citations: 1 2 1 1 1 6
Citation information
This citation data is accumulated from CrossRef, which receives citation information from participating publishers, including this journal. Not all publishers participate in CrossRef, so this information is not comprehensive. Additionally, data may not reflect the most current citations to this article, and the data may differ from citation information available from other sources (for example, Google Scholar, Web of Science, and Scopus).

Citations to this article (6)

Title and authors Publication Year
Podocyte Sphingolipid Signaling in Nephrotic Syndrome
Li G, Kidd J, Gehr TW, Li PL
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2021
Lipid biology of the podocyte—new perspectives offer new opportunities
A Fornoni, S Merscher, JB Kopp
Nature Reviews Nephrology 2014
Chronic kidney disease and an uncertain diagnosis of Fabry disease: Approach to a correct diagnosis
L der Tol, E Svarstad, A Ortiz, C Tøndel, JP Oliveira, L Vogt, S Waldek, DA Hughes, RH Lachmann, W Terryn, CE Hollak, S Florquin, MA van Weerman, C Wanner, ML West, M Biegstraaten, GE Linthorst
Molecular Genetics and Metabolism 2014
Phenotypical characterization of α-galactosidase A gene mutations identified in a large Fabry disease screening program in stroke in the young
ID Brabander, L Yperzeele, CC Groote, R Brouns, R Baker, S Belachew, J Delbecq, GD Keulenaer, S Dethy, F Eyskens, A Fumal, D Hemelsoet, D Hughes, S Jeangette, D Nuytten, P Redondo, B Sadzot, C Sindic, R Sheorajpanday, V Thijs, CV Broeckhoven, PP de Deyn
Clinical Neurology and Neurosurgery 2013
Activator protein required for the enzymatic hydrolysis of cerebroside sulfate. Deficiency in urine of patients affected with cerebroside sulfatase activator deficiency and identity with activators for the enzymatic hydrolysis of GM1 ganglioside and globotriaosylceramide
SC Li, H Kihara, S Serizawa, YT Li, AL Fluharty, JS Mayes, LJ Shapiro
The Journal of biological chemistry 1985
Increased urinary excretion of glycosphingolipids in familial hypercholesterolemia
S Chatterjee, CS Sekerke, PO Kwiterovich
Journal of lipid research 1982

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