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Abstract
For decades, peripartum cardiomyopathy has remained an enigma. Despite extensive research, our understanding of how a previously healthy woman can develop lethal heart failure in the context of pregnancy remains vague. Recent work suggests that inadequacy of the cardiac microvasculature may be the primary abnormality and has implicated an antiangiogenic fragment of the nursing hormone prolactin as playing an important role. In this issue of the
Authors
Ying Yang, Jessica E. Rodriguez, Richard N. Kitsis
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ISSN: 0021-9738 (print), 1558-8238 (online)