Abstract
Hyperaminoacidemia is a powerful stimulus of pancreatic glucagon secretion. These studies were designed to elucidate the role of aminogenic hyperglucagonemia in glucoregulation. Conscious dogs with previously implanted indwelling venous catheters were employed. The results support the view that a role of glucagon is to limit blood glucose decline during hyperaminoacidemia.
Authors
Roger H. Unger, Akira Ohneda, Eugenio Aguilar-Parada, Anna M. Eisentraut
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