[CITATION][C] Exercise-induced hypoglycemia following propranolol in a patient after gastric fundoplication surgery
GP Zaloga, RF Dons - Digestive diseases and sciences, 1984 - Springer
GP Zaloga, RF Dons
Digestive diseases and sciences, 1984•SpringerDISCUSSION Exercise requires major metabolic and cardiovascular adjustments in order to
increase the supply of fuels and oxygen to brain, muscle, and other organs. Normally the
blood glucose varies little despite marked increases in exercise-stimulated glucose
utilization (1, 2, 9). Normoglycemia is maintained by an increase in hepatic glucose output
(2, 9). Increases in catecholamine secretion during exercise (2, 7) decrease insulin secretion
via alphaadrenergic receptor stimulation and increase glucagon output via beta-adrenergic …
increase the supply of fuels and oxygen to brain, muscle, and other organs. Normally the
blood glucose varies little despite marked increases in exercise-stimulated glucose
utilization (1, 2, 9). Normoglycemia is maintained by an increase in hepatic glucose output
(2, 9). Increases in catecholamine secretion during exercise (2, 7) decrease insulin secretion
via alphaadrenergic receptor stimulation and increase glucagon output via beta-adrenergic …
DISCUSSION
Exercise requires major metabolic and cardiovascular adjustments in order to increase the supply of fuels and oxygen to brain, muscle, and other organs. Normally the blood glucose varies little despite marked increases in exercise-stimulated glucose utilization (1, 2, 9). Normoglycemia is maintained by an increase in hepatic glucose output (2, 9). Increases in catecholamine secretion during exercise (2, 7) decrease insulin secretion via alphaadrenergic receptor stimulation and increase glucagon output via beta-adrenergic receptor stimulation. These changes serve to increase glucose secretion by the liver. Beta2-adrenergic stimulation by catecholamines increases muscle cell glycogen-
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