Clues as to why long-lived species live so much longer than short-lived species may reside
in the amount of reactive oxygen species (ROS) produced and their effect on damaging cell
components (especially proteins) and alterations of crucial cellular processes. Rigorous
evaluation of these concepts required critical comparisons of oxidative damage markers
and/or parameters with assess difference in ROS flux and the critical age-modifying
processes they influence. The limited experimental comparative results available implicate …