Pre-eclampsia, a human disease of pregnancy, is the leading cause of maternal morbidity in
the western world. Despite the ubiquity of the disease and the plethora of studies concerning
its aetiology and pathogenesis no comprehensive theory concerning its aetiology and
pathogenesis have been put forward until now and so far there are no adequate therapies
other than bed rest and, if necessary, early delivery. We recently developed an animal
model for preeclampsia [1, 2], whereby activation of the inflammatory response by low dose …