Macrophages and Polymorphonuclear neutrophils in lung defense and Injury1-2
Y Sibille, HY Reynolds - Am Rev Respir Dis, 1990 - atsjournals.org
Y Sibille, HY Reynolds
Am Rev Respir Dis, 1990•atsjournals.orgThrough the release of numerous secretory products (table 1) and the expression of several
surface receptors (table 2), the macrophage can interact with other cells and molecules. This
strongly supports a central role for pulmonary macrophages. In the normal lung,
macrophages are resident cells, whereas PMN are almost absent. However, in certain
conditions, PMN can accumulate within the lung structures. The elucidation of the
mechanisms regulating PMN traffic from blood vessels to lung interstiti-
surface receptors (table 2), the macrophage can interact with other cells and molecules. This
strongly supports a central role for pulmonary macrophages. In the normal lung,
macrophages are resident cells, whereas PMN are almost absent. However, in certain
conditions, PMN can accumulate within the lung structures. The elucidation of the
mechanisms regulating PMN traffic from blood vessels to lung interstiti-
Through the release of numerous secretory products (table 1) and the expression of several surface receptors (table 2), the macrophage can interact with other cells and molecules. This strongly supports a central role for pulmonary macrophages. In the normal lung, macrophages are resident cells, whereas PMN are almost absent. However, in certain conditions, PMN can accumulate within the lung structures. The elucidation of the mechanisms regulating PMN traffic from blood vessels to lung interstiti-
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