The role of cerebral amyloid β accumulation in common forms of Alzheimer disease
J. Clin. Invest. Sam Gandy, et al. 115:1121 doi:10.1172/JCI25100 [Go to this article.]

Figure 4
(A) Structure and topology of APP. (B) The fine structure around the Aβ domain, secretase cleavage sites, and locations of some selected familial AD missense mutations. Figure modified with permission from Trends in Endocrinology and Metabolism: TEM (15).