The adipocyte fatty acid–binding protein aP2 is required in allergic airway inflammation
J. Clin. Invest. Bennett O.V. Shum, et al. 116:2183 doi:10.1172/JCI24767 [Go to this article.]

Figure 4
Evidence for the role of aP2 in Th2-mediated airway inflammation (A) HBEs were treated with a panel of cytokines and stimuli and aP2 expression assessed with real-time PCR. Data represent mean ± SEM of 3 experiments for IL-4, IL-13, and IFN-γ; 1 experiment for IL-9 and MCP-1; and 2 experiments for the other treatments. (B) BALB/c mice in which allergic lung inflammation was induced or control mice were immunostained for aP2: brown, positive aP2 staining; blue, nuclei. Images are representative of 3 mice per condition. Original magnification, ×400.