FGF-2 controls the differentiation of resident cardiac precursors into functional cardiomyocytes
J. Clin. Invest. Nathalie Rosenblatt-Velin, et al. 115:1724 doi:10.1172/JCI23418 [Go to this article.]

Figure 7
Cell transfer into neonatal mouse recipients. (A) PKH2-GL–labeled cells or cells constitutively expressing the GFP gene under the chicken β-actin promoter (GFP) were injected i.v. into neonatal mice. Ten days thereafter, heart sections were stained for α-actinin. (B) PKH2-GL–labeled FGF-2^+/+ or FGF-2^–/– NMCs were injected into neonatal FGF-2^+/+ or FGF-2^–/– mice. Ten days after injection, heart sections were stained for α-actinin. (C) WT PKH2-GL–labeled NMCs were injected into neonatal ROSA26 transgenic mice, and heart sections were stained for β-gal. Original magnification, ×63 (A and C), ×20 (B).