AMP-activated protein kinase mediates ischemic glucose uptake and prevents postischemic cardiac dysfunction, apoptosis, and injury
J. Clin. Invest. Raymond R. Russell, et al. 114:495 doi:10.1172/JCI19297 [Go to this article.]

Figure 3
α2- and α1-specific AMPK activity from hearts from KD and WT mice after baseline perfusion period (n = 3 for both), after ischemia (n = 4 for both), and following 30 minutes of ischemia and 30 minutes of reperfusion (n = 7 for WT and n = 6 for KD). *P < 0.01 versus baseline; ^† P < 0.01 versus WT.