Constitutively active AMP kinase mutations cause glycogen storage disease mimicking hypertrophic cardiomyopathy
J. Clin. Invest. Michael Arad, et al. 109:357 doi:10.1172/JCI14571 [Go to this article.]

Figure 3
Snf1-Snf4 two-hybrid interactions in the presence and absence of glucose. β-Galactosidase activity was measured in cell cultures grown in SD glucose (a) or galactose/raffinose (b) media. The activity from three cultures from different colonies is reported in units (hydrolysis of 1 μmol CPRG/min/cell), mean ± SD. WT, wild-type. *P < 0.02, **P < 0.005 versus wild-type Snf4.